Monday, 22 September 2014

ACLR Rehab and Return to Sport Criteria


ACL Rehab & Return to Sport Criteria

This blog entry is based on an assignment on ACL return to sport criteria from my Masters course last year (thanks must go to my peers, especially Kylie Baldwin and Jennifer Callaghan) as well as information from APA Sports Physiotherapist Randall Cooper and his recent presentation on ACL Rehab Criteria. 

Randall presented a 5 stage criteria driven rehab protocol including the Melbourne Sports Score to assist in progressing rehab following ACL reconstruction and knowing when a patient is safe and ready to return to sport.  I’ll discuss each stage briefly with my opinions and variations, but you can find the full documents describing each stage on Randall’s blog here: www.swollenknee.me Thanks again to Randall for being so generous with sharing this information.

I’d love to hear any variations or additional criteria that you use, or recommend using.

Stage 1: Immediate Post Op – Recovery from Surgery

Aims
Treatment Options
Criteria to Exit Stage 1
Reduce pain
Ice
Full PROM Knee Extension
Reduce swelling
Elevation
PROM Knee Flexion 100deg
Safe mobilisation
Pain relief
Zero to mild amount of swelling
 
Adequate rest
Quads lag 0-5deg
 
Braces, crutches as needed
 

Goals of this first stage are to regain knee extension, settle the swelling and activate the quads.

Stage 2: Strength and Neuromuscular Control

Aims
Rehab
Criteria to Exit Stage 2
Regain single leg balance
Massage
Full PROM Knee Extension
Regain muscle strength
Joint mobilisations
125deg+ PROM Knee Flexion
Good single leg squat technique and LL alignment
Quads (contracted with hamstrings) strengthening – IRQ, mini squats, squats, mini lunges, lunges, leg press double, leg press single
No swelling or effusion
 
Hamstring Strengthening – bridging, deadlifts, prone or seated hamstring curls, hamstring flicks
5 single leg squats with ‘good’ alignment based on Crossley et al 2011
 
Calf Strengthening – calf raises
No. single leg bridges >85% of other leg
 
Abdominal/Core strengthening – plank, side plank, bridge and leg lift, leg lifts, leg slides/extensions, crunches
No. single leg calf raises >85% of other leg
 
Ice/elevation as needed – mostly after exercise sessions
Timed side bridge/plank >85% of other side
 
Cross training UL
1RM single leg press = 1.5xbody weight
 
Ex bike, rowing for cardio
Single leg balance eyes open=43sec, eyes closed=9sec

 

So here’s where we start to debate the numbers.  Are 5 single leg squats enough to prove you have sufficient neuromuscular control to move to Stage 3?  If your athlete is returning to a running sport, then they will eventually need to squat/hop/jump a lot more than 5 times.  Crossley’s test is also based on patellofemoral pain, so we need to decide whether this can be extrapolated to the ACLR population – time for another RCT.  Another question is whether 1.5x body weight is too much (or too little) as a Strength measure in Stage 2.  We also need to remember when comparing % of one leg to another that the so called ‘good’ leg may have previously been injured and thus the comparison is not entirely valid. 

Stage 3: Running, Agility and Landing

Aim
Rehab
Criteria to Exit Stage 3
‘excellent’ score on jump-rebound task
Shuttle runs
Single Leg Hop test >90% of other leg
Progressive agility training
Ladder drills
Triple Cross Over Hop Test >90% of other leg
Regain full Strength and Balance
Slalom running/cutting drills
Landing Error Scoring System (LESS) “Excellent” Score
 
Jumping – double leg, floor, box, perturbations
1RM Single leg press 1.8x body weight
 
Hopping – single leg, floor, box, change of direction
Star Excursion Balance Test (SEBT) >95% of other leg in Anterior, Posterolateral and Posteromedial directions

The patient must have met the criteria of Stage 1 and 2 before entering this Stage of rehab.  I can’t justify starting a patient on a return to running program if they only do 5 single leg squats.  My general outcome measures for returning to running are 30 single leg squats, 30 single leg calf raises and 30 single leg bridges as a minimum.  I’d be interested to hear what your criteria are!?

Before testing the exit criteria for Stage 3, it is also recommended that the patient have no side to side difference in the single leg bridge test, single leg calf raise test and side bridge endurance test, while maintaining FROM in the knee, no swelling and ‘good’ single leg squats.  

Stage 4:Return to Sport

Aim
Rehab
Criteria to Return to Sport
(Melbourne Sports Score)
Score >95 on Melbourne RTS Score
Sports Specific Drills/Skills
Strength and Conditioning Program
Physical Examination (/25)
Effusion
Stability/Laxity
Flexion ROM
Extension ROM
Athlete is confident, comfortable and eager to RTS
Plyometrics
Functional Tests (/50)
SEBT
Single Hop for distance
Triple Hop
Jump/Land ability – modified LESS score
SL Squats
A continued ACL injury prevention program is discussed and implemented.
PEP or FIFA 11 prevention program (stage 5)
IKDC/Subjective Qs (/25)

 

Should we be including more tests?

Are these criteria enough to justify safe RTS? Pilot data suggests that a score of 95/100 indicates a greater chance of returning to pre-injury sports.  Further research is also needed to see whether these athletes (scoring >95/100) also have a lower re-injury rate.

Stage 5: Prevention

There are a number of preventative programs already available, including FIFA 11 and PEP program.  You can find these online easily.

Conservative Management

Apart from the acute post op management, conservatively management ACL injuries should follow a similar set of rehab and return to sport criteria. 

Thursday, 20 February 2014

Anterior Cruciate Ligments (ACLs)

With the winter Olympics almost finished, we've been hearing about anterior cruciate ligment (ACLs) injuries every day, so I thought it timely to discuss this in a bit more detail.

The ACL originates on the anterior intercondylar area of the tibia and inserts on the posteromedial aspect of the lateral femoral condyle.  It is composed of two bundles of type 1 collagen fibres; an anteromedial bundle (taught in flexion) and a posterolateral bundle (taught in extension).  The ACL is the primary restraint to anterior translation of the tibia relative to the femur.  It plays a secondary role in restraining internal rotation particularly when the knee is close to full extension.     




A number of risk factors have been associated with ACL injuries.  They can be divided into 4 categories:

Anatomical

-          Increased BMI

-          Narrow femoral notch width

-          Greater ligament laxity

-          Wider pelvis and Q angle

Hormonal

-          Oestrogen levels during early and late follicular phase of menstrual cycle

Environmental

-          Harder surfaces

-          Increased shoe-surface friction

-          Cleat position

Neuromuscular 

-          Poor ability to control dynamic knee valgus

-          Quads/hamstring ratio

-          Reduced knee and knee flexion angles

Previous ACL injury is also a risk factor for ACL injury for both the reconstructed or contralateral knee (Orchard et al 2001).

Approximately 70% of ACL injuries are non-contact injuries, usually seen in sports and activities involving complex movements such as cutting and pivoting account (e.g. netball, football) (Hernandez, 2006).  A non-contact ACL rupture usually involves a rotational or lateral valgus force on the knee with the knee flexed and the foot planted.  An example of this is the football player who steps off his foot in a cutting manoeuvre to try and change direction, only to have his knee buckle underneath him   http://www.youtube.com/watch?v=lpIOMuqXWrE Contact ACL injuries often occur from hyperextenion or valgus forces applied externally to the knee (e.g. contact from an opponent).

Patients often report hearing a ‘pop’ as the ACL ruptures.  Gross swelling of the knee and spasm of the hamstrings usually occurs within minutes.  Standard assessment involves a stress test for the ACL – these include Lachman’s, pivot shift and anterior drawer.  Unless performed soon after injury, hamstring spasm can limit the effectiveness of these tests.  Patients report pain with knee movement, decreased ROM and instability of their knee.   MRI is the current gold standard for diagnostic imaging.  This is also useful to investigate other pathology that may be present such as medial meniscus injury or MCL tear (unhappy triad).  Whether you can have a partial tear of the ACL also remains debatable.    

 
 

Pictures from: http://www.emedx.com/emedx/diagnosis_information/knee_disorders/acl_mri_pictures.htm

ACL tears do not heal after injury.  Stitching the ligament back together has generally failed due to a poor blood supply and poor healing environment created by the synovial fluid.  As a result, ACL injuries are costly in a number of ways: time lost from sport, financial costs of rehab and/or surgical repair, long term consequences such as osteoarthritis, and decreased quality of life.  
Which brings up the questions: Can you function without an ACL and what are the consequences of doing so?

We know the ACL helps with dynamic stability of the knee so without an ACL the stability of the knee is compromised which potentially MAY cause further damage such as meniscus tears.  However in some patients, this can be compensated for with good neuromuscular control.

Eitzen et al (2010) recommend a period of rehab (prehab) to inform decision making for the management of ACL injury (ie conservative vs surgical).  And what a great idea that is (no sarcasm intended)!  As a sports physio I often see patients with ACL tears who almost immediately see a surgeon who operates within days to weeks without any form of prehab.  I don’t think this is ideal for a number of reasons including: a) some patients can function without an ACL, b) prehab results in better outcomes post-surgery and c) patients can have a false sense of security that after surgery their knee will be just like new. 

Prehab according to Eitzen’s (2010) study should start when initial impairments have resolved (no joint effusion, no gait abnormalities, equal ROM, able to hop pain free).  Whether this is suitable criteria remains to be tested.  I would think if you can hop pain free you’re doing ok.  Initial testing involved 4 single leg hop tests, self-reported questionnaires, global rating of knee function, episodes of giving way and isokinetic quads and hammy strength tests.  Eitzen’s prehab program then involved progressive exercise therapy for 5 weeks (for patients playing level 1 and 2 sports – see table below) followed by retesting.    Results from this study found a significant improvement in knee function in patients with ACL injury suggesting a period of prehab is important prior to surgery or as the beginning of a progressive conservative rehab program. 

Activity Level & Skill Requirements

Level
Skills
Example Sports
1
Jumping, Cutting, Pivoting
Soccer, Netball, Football, Basketball
2
Lateral Movement OR
Heavy manual labour OR working on uneven surfaces
Tennis, Skiing
3
Light activity
Running, weight lifting
4
Sedentary activity
Housework, ADLs

 

Frobell et al (2010, 2013) have shown that delaying reconstruction does not affect outcomes.  Two year and 5 year data show patients (active, but not elite athletes) who had early reconstruction (<10wks) did not have better self-reported outcomes (KOOS) than those who had delayed reconstructions (>5-19mths).  As physiotherapists we should be encouraging a period of prehab prior to any surgery.  This will always be influenced by the athlete, level of sport they play, demands of other parties (club, coach, sponsors, family), but it is important to inform the athlete that prehab does improve knee function. 

Research is finally coming out comparing the long term outcomes of patients with ACL injuries that did and didn’t have reconstructive surgery.  Ageberg et al (2008) found no difference in muscle strength, functional performance (hop tests) or limb symmetry at 2-5 year follow up.  Studies by Meuffels et al (2009) and Streich et al (2011) also showed no significant difference in functional outcomes or activities levels at 10 and 15 year follow ups respectively.  Another recent study from Lynch et al (2013) did show a significantly higher rate of return to level 1 sport in patients with reconstructed ACLs.  It is important to note that patients who had conservative rehab where advised not to return to level 1 sports. 

For years surgeons and physios have advised patients to have their ACL reconstructed to minimise the risk of osteoarthritis (OA) and further damage to the knee joint.  We now have some evidence (read above studies) to suggest this may not be true and the literature is unclear at this point in time whether ACL reconstruction does reduce OA and meniscus injury rates. 

So where does that leave us? Hopefully this flow chart will help you and your patient make an informed decision:
 
 
 
In my next post I’m going to discuss what should be included in prehab, rehab and when to return to sport.  Questions, comments most welcome on this topic!
LB
 
 
References:
 
 
 

Thursday, 6 February 2014

Scapula Dyskinesia


The shoulder blade or scapula (2 scapulae) is crucial to how the shoulder functions, but also plays a large role in our upper body posture and can contribute to neck pain.  Triangular in shape, the scapula lies against the back of the rib cage.  Anteriorly it is connected to the chest via the collar bone (clavicle). 

 

The scapula can move in a number of planes allowing several different movements to occur:

·         Elevation and depression

·         Upward and downward rotation

·         Protraction and retraction

·         Anterior and posterior tilt


 
Getting your head around these movements is important for assessing scapula posture at rest and under load.  Muscle balance around the shoulder blade, neck and thoracic spine effects the position of the scapula and thus the stability and mobility of the neck and shoulder.  17 different muscles attach to the scapula. 

  1. Serratus Anterior
  2. Supraspinatus
  3. Subscapularis
  4. Trapezius
  5. Teres Major
  6. Teres Minor
  7. Triceps Brachii long head
  8. Biceps Brachii
  9. Rhomboid Major
  10. Rhomboid Minor
  11. Coracobrachialis
  12. Omohyoid inferior belly
  13. Lattisimus Dorsi
  14. Deltoid
  15. Levator Scapula
  16. Infraspinatus
  17. Pectoralis Minor

Having an appropriate group of muscles working around the scapula (force couple) enables the scapula to work effectively as a base for the shoulder to move from and limits excess strain on the neck.  An imbalance in these muscles often results in what is called scapula dyskinesis (a fancy name for a movement disorder of the shoulder blade).      

In the diagram below you can see the different pull of 3 muscles (lower traps, upper traps and serratus anterior) all attaching to the scapula.  This force couple is particularly important when the shoulder is moving and we see what is called scapulohumeral rhythm. 

Picture from: http://nateregensburg.blogspot.com.au/2010/12/increasing-upper-body-strength-by.html   

Today I particularly want to discuss the neutral position of the scapula when the arm is in neutral (by the side of the body).  You might ask why is that important when you don’t play sport with your arm held by your side?  We do, however, spend a lot of our day with arm/s close to our body – walking, working at a desk or computer, eating, sitting on the couch.  For athletes it’s important that they don’t have poor posture during the day, leading to muscle imbalance, before going to training or competition, as this can adversely affect their performance or cause injury     

The neutral position of the scapula is flat against the ribcage with no rotation, tilt or elevation present.   The medial border of the scapula should be vertical.    Landmarks commonly used to assess scapula position are the spine of the scapula at T3 and the inferior angle of the scapula level with T7.  This is not entirely accurate as the size of each scapula will vary depending on the size of the person. 


 
Commonly patients with neck and/or shoulder pain present to physiotherapy and winging scapula/e are noted on assessment.  Winging occurs when the medial (inside) border of the scapula lifts away from the ribcage.  We should consider 2 types and causes of winging:

1) Winging of the entire medial border – due to weakness in serratus anterior

2) Winging of the inferior angle of the scapula – due to weakness in lower trapezius

If you suspect weakness in serratus anterior it’s important to rule out a long thoracic nerve palsy.  This can occur from trauma or infection or be insidious.  It requires a longer period of rehab and referral for medical opinion. 

Both types of winging and other forms of scapula dyskinesia (e.g. downwardly rotated scapula at rest) require postural correction and exercise prescription. 

Far too often physios (and pilates instructors and other health professionals), cue people to pull their shoulder blades back and down to set a neutral scapula position.  The problem here is not all shoulder blades are sitting forward and up, so this cue to set the scapula is not going to correct all scapulae properly.  Having a generalised, non specific cue like “draw down and back” leads to problems. 

Remembering that the role of lower traps is to depress and upwardly rotate the scapula we need to have more specific cues for patients with scapula dyskinesia.

Highlighted below is the right levator scapulae muscle.  On the left, the overlying trapezius muscle is still present.  Levator scapulae elelvates the scapula, but also downwardly rotates the scapula (blue arrow) in combination with other muscles such as pec minor. This downward rotation is counterbalanced by the fibres of upper traps and lower traps (green arrow) with lower traps also balancing the elevation of the scapula.  The force couple between these muscles needs to work to enable a neutral scapula position.   


There’s no recipe I can give you for treating scapula dyskinesia and each patient will respond differently to different cues.  If you take the time to assess the scapula position properly and then base your postural cueing and exercise program on this I don’t think you can go wrong.  Tactile (palpation, sweep tapping, taping)  and visual (computer biofeedback) also help heaps!!

LB

 

Some other reading you might be interested in: Link here to a recent paper from scapula guru Ben Kibler and colleagues: Clinical implications of scapular dyskinesis in shoulder  injury: the 2013 consensus statement from the ‘scapular summit ’http://bjsm.bmj.com/content/47/14/877.full.pdf+html

 














 
 
 
 
 
 
 
 
 
 
 
 
 

 
 
 
 
 
 
 
 











 

 
 

 

 
 

 

 
 

 

 
 

 

 
 

 

 
 

 

 
 





 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
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